Research Article TRAF1/C5 but Not PTPRC Variants Are Potential Predictors of Rheumatoid Arthritis Response to Anti-Tumor Necrosis Factor Therapy

نویسندگان

  • Helena Canhão
  • Ana Maria Rodrigues
  • Maria José Santos
  • Diana Carmona-Fernandes
  • Bruno F. Bettencourt
  • Jing Cui
  • Fabiana L. Rocha
  • José Canas Silva
  • Joaquim Polido-Pereira
  • José Alberto Pereira Silva
  • José António Costa
  • Domingos Araujo
  • Cândida Silva
  • Helena Santos
  • Cátia Duarte
  • Rafael Cáliz
  • Ileana Filipescu
  • Fernando Pimentel-Santos
  • Jaime Branco
  • Juan Sainz
  • Robert M. Plenge
  • Daniel H. Solomon
  • Jácome Bruges-Armas
  • José António P. Da Silva
  • João Eurico Fonseca
چکیده

Helena Canhão, Ana Maria Rodrigues, Maria José Santos, Diana Carmona-Fernandes, Bruno F. Bettencourt, Jing Cui, Fabiana L. Rocha, José Canas Silva, Joaquim Polido-Pereira, José Alberto Pereira Silva, José António Costa, Domingos Araujo, Cândida Silva, Helena Santos, Cátia Duarte, Rafael Cáliz, Ileana Filipescu, Fernando Pimentel-Santos, Jaime Branco, Juan Sainz, Robert M. Plenge, Daniel H. Solomon, Jácome Bruges-Armas, José António P. Da Silva, João Eurico Fonseca, and ElizabethW. Karlson

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منابع مشابه

TRAF1/C5 but Not PTPRC Variants Are Potential Predictors of Rheumatoid Arthritis Response to Anti-Tumor Necrosis Factor Therapy

BACKGROUND The aim of our work was to replicate, in a Southern European population, the association reported in Northern populations between PTPRC locus and response to anti-tumor necrosis factor (anti-TNF) treatment in rheumatoid arthritis (RA). We also looked at associations between five RA risk alleles and treatment response. METHODS We evaluated associations between anti-TNF treatment res...

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Lack of Association of Variants Previously Associated with Anti-TNF Medication Response in Rheumatoid Arthritis Patients: Results from a Homogeneous Greek Population

Treatment strategies blocking tumor necrosis factor (anti-TNF) have proven very successful in patients with rheumatoid arthritis (RA), showing beneficial effects in approximately 50-60% of the patients. However, a significant subset of patients does not respond to anti-TNF agents, for reasons that are still unknown. The aim of this study was to validate five single nucleotide polymorphisms (SNP...

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Replication of Association of the PTPRC Gene With Response to Anti–Tumor Necrosis Factor Therapy in a Large UK Cohort

OBJECTIVE Several rheumatoid arthritis (RA) susceptibility variants map close to genes involved in the tumor necrosis factor (TNF) signaling pathway, prompting the investigation of RA susceptibility variants in studies of predictors of response to TNF blockade. Based on a previously reported association of RA with the PTPRC genetic locus, the present study was undertaken to test established RA ...

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TRAF1-C5 as a risk locus for rheumatoid arthritis--a genomewide study.

BACKGROUND Rheumatoid arthritis has a complex mode of inheritance. Although HLA-DRB1 and PTPN22 are well-established susceptibility loci, other genes that confer a modest level of risk have been identified recently. We carried out a genomewide association analysis to identify additional genetic loci associated with an increased risk of rheumatoid arthritis. METHODS We genotyped 317,503 single...

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High incidence of cardiovascular events in patients with rheumatoid arthritis.

1. Abe M. Complement activation and inflammation. Rinsho Byori 2006;54:744–56. 2. Plenge RM, Seielstad M, Padyukov L, Lee AT, Remmers EF, Ding B, et al. TRAF1-C5 as a risk locus for rheumatoid arthritis – a genomewide Study. N Engl J Med 2007;357:1199–209. 3. Kurreeman FA, Padyukov L, Marques RB, Schordi SJ, Seddighzadeh M, StoekenRijsbergen G, et al. A candidate gene approach identifies the TR...

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تاریخ انتشار 2015